2'-AA

2'-Acetylacteoside has antioxidative activity. It can significantly attenuate glutamate-induced neurotoxicity at concentrations ranging from 0.1 to 10 μM. 2'-Acetylacteoside shows antiproliferative effects on aortic smooth muscle, indicates that it may ha

Norepinephrine is an alkaloid neurotransmitter and an effective adrenergic receptor (AR) agonist that activates α1, α2, and β1 receptors. It is commonly used as a vasoactive agent for the treatment of shock and can also be used to induce cardiomyopathy models.

Benzylamine hydrochloride, made of benzylamine and hydrochloric acid, is used to treat motion sickness and ascariasis, with antiemesis properties.

3′-Methoxyfurano[4″,5″:3,4]chalcone (Compound 2) is a selective A2AAR antagonist with an IC50 of 33.5 nM, indicating it has a high affinity. This compound is a natural product that can be extracted from the skins of Allium cepa L. Additionally, 3′-Methoxyfurano[4″,5″:3,4]chalcone is capable of promoting T cell activation and is useful in cancer immunology research.

2-O-β-D-Glucopyranosyl-L-ascorbic acid (AA-2βG) is a natural and stable vitamin C analog that induces apoptosis and cell cycle arrest in cancer cells by stabilizing p53 protein.

1.Aristolactam I (Aristololactum) has cytotoxic potency, mediated through the induction of apoptosis in a caspase 3-dependent pathway. 2. The effects of Aristololactam I may be mediated by different mechanisms except TGF-beta1 pathway. 3. Aristololactam I may be one of toxic metabolites in Chinese herbs containing AA which participate in renal damage and fibrosis.

Bactenecin TFA (Bactenecin, bovine TFA), a potent 12-aa looped antimicrobial peptide derived from bovine neutrophils, effectively inhibits bacterial and yeast growth, eradicates Trichophyton rubrum fungus, enhances membrane permeability, and suppresses B. pseudomallei growth and biofilm formation [1] [2] [3].

Aristolactam I (Standard) is a reference standard for research and analysis in studies involving Aristolactam I. 1.Aristolactam I (Aristololactum) has cytotoxic potency, mediated through the induction of apoptosis in a caspase 3-dependent pathway. 2. The effects of Aristololactam I may be mediated by different mechanisms except TGF-beta1 pathway. 3. Aristololactam I may be one of toxic metabolites in Chinese herbs containing AA which participate in renal damage and fibrosis.

2-Deacetoxytaxinine B is a strongly inhibitor against U46619-induced aggregation, it shows strong inhibitory effects against arachidonic acid (AA)-induced aggregation.