gln

Memogain is a pro-drug of galantamine. It is used for the treatment of Alzheimer's disease.

Gln-AMS is an inhibitor of aminoacyl-tRNA synthetases (AARS). It specifically binds to the A-domain located within the NRPS enzymes.

Gln-AMS(TFA) inhibits glutaminyl-tRNA synthetase (GlnRS) with a Ki of 1.32 μM. Gln-AMS (TFA) is a type Ia aminoacyl-tRNA synthetase (AARS) inhibitor.

Fmoc-Gln(Trt)-Thr(psi(Me,Me)pro)-OH is a dipeptide.

Fmoc-Gln(Trt)-Ser(psi(Me,Me)pro)-OH is a dipeptide.

Cyclo(Gly-Gln) is a cyclic dipeptide.

[Gln8] Luteinizing hormone releasing hormone ([Gln8] LH-RH), Avian hypothalamic peptide that stimulates release of gonadotropins from anterior pituitary, thus regulating reproductive functions.

[Gln8]-C517 (LH-RH), chicken acetate is an avian hypothalamic peptide that stimulates release of gonadotropins from anterior pituitary, thus regulating reproductive functions.

An endogenous ligand for the ghrelin receptor (GHS-R1a), produced by alternative splicing of the rat ghrelin gene. Potently induces Ca2+ release in cells expressing ghrelin receptors (EC50 = 2.4 nM) and stimulates GH release in vivo.

pGlu-Gln-Arg-Leu-Gly-Asn-Gln-Trp-Ala-Val is a peptides

GLN51108 is a synthetic porphyrin derivative primarily used to initiate artificial photosynthesis and induce apoptosis. Additionally, GLN51108 is also employed in catalytic heterogeneous oxidation.

H-γ-Glu-Gln-OH (γ-L-glutamyl-L-glutamine) is a highly hydrophilic dipeptide utilized as a building block for advanced drug delivery systems. Carriers composed of H-γ-Glu-Gln-OH, such as poly(L-γ-glutamylglutamine) (PGG), are characterized by high water solubility, superior drug-loading capacity, excellent biocompatibility, and low systemic toxicity. Conjugating drugs (e.g., Paclitaxel) to this peptide moiety significantly improves the solubility of hydrophobic agents and enhances tumor-targeting and tissue penetration (especially in glioblastoma models), leading to optimized anti-tumor efficacy.

GPNA hydrochloride is specific glutamine (Gln) transporter ASCT2(SLC1A5) inhibitor. GPNA reversibly induces apoptosis in A549 cells. GPNA hydrochloride is a well-known substrate of the enzyme γ-glutamyltransferase (GGT). GPNA hydrochloride also inhibits

Phenylacetylglutamine (NSC 203800)is a normal constituent of human urine, but other mammals such as the dog, cat, rat, monkey, sheep, and horse do not excrete this compound. Phenylacetylglutamine is a major nitrogenous metabolite that accumulates in uremia. Phenylacetylglutamine also is a microbial metabolite found in Christensenellaceae, Lachnospiraceae, and Ruminococcaceae.

L-Alanyl-L-glutamine (Ala-Gln), a dipeptide composed of alanine and Gln, is an alternative supplement to L-glutamine in the production of biopharmaceuticals with better solubility and stability than glutamine (Gln).

Ac-DEVD-AMC (AC-ASP-MET-GLN-ASP-7-AMINO-4-METHYLCOUMARIN) is the substrate of Caspase-3 .

Levitide is a neurohormone-like peptide which is from the skin of Xenopus laevis.

R 396 (Acleu-asp-gln-trp-phe-glynh2) is a linear hexapeptide and is also an NK-2 tachykinin receptor antagonist.

alpha-Gliadin (43-49) is a Gliadian sequence peptide. It could induce leukocyte migration inhibition but be blocked by naloxone.

Felypressin (Octapressin) is a synthetic analog of LYPRESSIN with a PHENYLALANINE substitution at residue 2. Felypressin is a vasoconstrictor with reduced antidiuretic activity.

Felypressin acetate (PLV-2) is an agonist of vasopressin 1 and acts on all arginine vasopressin receptors 1AS. Felypressin acetate can be used in dental procedures.

N-Butoxyacetylglutamine is a metabolite of N-butoxyacetic acid.

Pro-Adrenomedullin(153-185),human, (C143H224N42O43), a peptide with the sequence H2N-SLPEAGPGRTLVSSKPQAHGAPAPPSGSAPHFL-OH, MW= 3219.6. Adrenomedullin (AM) is a ubiquitously expressed peptide initially isolated from phaechromyctoma in 19931. AM was initially identified as a vasodilator, some have cited this as the most potent endogenous vasodilatory peptide found in the body2. Differences in opinion regarding the ability of AM to relax vascular tone arises from the differences in the model system used3. Other effects of AM include increasing the tolerance of cells to oxidative stress and hypoxic injury and angiogenesis. AM is seen as a positive influence in diseases such as hypertension, myocardial infarction, chronic obstructive pulmonary disease and other cardiovascular diseases, whereas it can be seen as a negative factor in potentiating the potential of cancerous cells to extend their blood supply and cause cell proliferation.

Nalpha-Fmoc-L-Glutamine (Fmoc-Gln-OH) is a glutamine derivative.