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(7)
By Tag- N-GST
- N-6xHis-SUMO(1)
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By Tag By Expression System- E. coli(8)
By Species- Human(5)
- Staphylococcus aureus(2)
- Rat(1)
By SPR-compatible Buffer- Buffer-exchangeable(6)
- Ready-to-use(2)
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Results for "
gst-b
" in TargetMol Product Catalog- Inhibitors & Agonists3
- Recombinant Protein8
- Antibody Products2
CompareCTSB, CPSB, Cathepsin B1, Cathepsin B, APP secretase (APPS)
TMPH-01059
Expression system: E. coli
Length: 82-333, Partial
Activity: Not Tested
Length: 82-333, Partial
Activity: Not Tested
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Buffer-exchangeable
ComparePreproprotein L7 (hPPL7), PPNPB, PPL7, NPB, Neuropeptide B
TMPH-01764
Expression system: E. coli
Length: 25-53, Full Length of Mature Protein
Activity: Not Tested
Length: 25-53, Full Length of Mature Protein
Activity: Not Tested
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Buffer-exchangeable
CompareSEB, Enterotoxin type B, entB
TMPH-03540
Expression system: E. coli
Length: 28-266, Full Length of Mature Protein
Activity: Not Tested
Length: 28-266, Full Length of Mature Protein
Activity: Not Tested
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Buffer-exchangeable
CompareStaphylopain B, Staphylococcal cysteine proteinase B, Staphopain B, sspB
TMPH-03577
Expression system: E. coli
Length: 220-393, Full Length of Mature Protein
Activity: Not Tested
Length: 220-393, Full Length of Mature Protein
Activity: Not Tested
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Buffer-exchangeable
CompareTranscription elongation factor B polypeptide 2, TCEB2, SIII p18, RNA polymerase II transcription factor SIII subunit B, Elongin-B, Elongin 18 kDa subunit, EloB, ELOB
TMPH-01288
Expression system: E. coli
Length: 1-118, Full Length
Activity: Not Tested
Length: 1-118, Full Length
Activity: Not Tested
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Buffer-exchangeable
Comparealdolase B, fructose-bisphosphate, ALDO2, ALDB
TMPY-02622
The aldolase family members involved in metabolism and glycolysis are present in three isoforms: ALDOA, ALDOB, and ALDOC. Aldolases are differentially expressed in human tissues, and aberrant expression has been observed in several human diseases and cancer types. Via GATA6, metastatic cells in the liver upregulate the enzyme aldolase B (ALDOB), which enhances fructose metabolism and provides fuel for major pathways of central carbon metabolism during tumor cell proliferation. Targeting ALDOB or reducing dietary fructose significantly reduces liver metastatic growth but has little effect on the primary tumor. Hereditary fructose intolerance (HFI) is an autosomal recessive disorder caused by aldolase B (ALDOB) deficiency resulting in an inability to metabolize fructose. The toxic accumulation of intermediate fructose-1-phosphate causes multiple metabolic disturbances, including postprandial hypoglycemia, lactic acidosis, electrolyte disturbance, and liver kidney dysfunction.
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SPR-compatible buffer
CompareLigandin, Gsta1, GST B, GST A1-1, GST 1a-1a, GST 1-1, Glutathione S-transferase Ya-1 (GST Ya1), Glutathione S-transferase alpha-1, Androst-5-ene-3,17-dione isomerase, 13-hydroperoxyoctadecadienoate peroxidase
TMPH-03305
Expression system: E. coli
Length: 2-222, Full Length of Mature Protein
Activity: Not Tested
Length: 2-222, Full Length of Mature Protein
Activity: Not Tested
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Buffer-exchangeable
CompareMGC75043, FLJ43543, FLJ22950, CD24A, CD24 molecule, CD 24
TMPK-00004
Expression system: E. coli
Length: 27-59, Full Length of Mature Protein
Activity: ELISA
Length: 27-59, Full Length of Mature Protein
Activity: ELISA
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SPR-compatible buffer
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