ursodeoxycholate

Ursodeoxycholic acid sodium (Sodium Ursodeoxycholate) is a naturally occurring secondary bile acid with anti-inflammatory and cytoprotective activities. Ursodeoxycholic acid sodium acts as signaling molecule, exerting its effects by interacting with bile acid activated receptors,

Ursodeoxycholic acid (UDCA) is a potent inhibitor of liver-specific fatty acid transporter 5 (FATP5). Ursodeoxycholic acid inhibits cholesterol absorption and is used to dissolve gallstones.

Berberine ursodeoxycholate (HTD1801) is an orally effective hypolipidemic agent, an ionic salt of Berberine and Ursodeoxycholic acid.Berberine ursodeoxycholate has a wide range of metabolic activity and significantly reduces liver fat content. Berberine ursodeoxycholate has been used in studies of hyperlipidemia, non-alcoholic steatohepatitis (NASH) and diabetes.

Tauroursodeoxycholate (UR 906), also known as ursodoxicoltaurine, is a highly hydrophilic tertiary bile acid that is produced in humans at a low concentration. Tauroursodeoxycholate is the more hydrophilic form of ursodeoxycholic acid, which is the more abundant naturally produced bile acid in humans.Tauroursodeoxycholate is being investigated for use in several conditions such as Primary Biliary Cirrhosis (PBC), insulin resistance, amyloidosis, Cystic Fibrosis, Cholestasis, and Amyotrophic Lateral Sclerosis.

Tauroursodeoxycholate sodium (TUDC) is an endoplasmic reticulum (ER) stress inhibitor, used for the treatment of gallstones and liver cirrhosis.

Tauroursodeoxycholate (TUDCA ,Taurolite) dihydrate is a water-soluble bile acid with endoplasmic reticulum stress inhibitory, mitochondrial stabilizing, and anti-apoptotic effects, which significantly reduces the expression of apoptotic molecules, such as Caspase-3 and Caspase-12, and inhibits ERK, reducing endoplasmic reticulum stress mediated cell death.

Tauroursodeoxycholate-D4 sodium is the deuterated form of Tauroursodeoxycholate sodium. Tauroursodeoxycholate (Tauroursodeoxycholic acid; TUDCA) sodium functions as an endoplasmic reticulum stress inhibitor. It significantly reduces the expression of apoptotic molecules such as caspase-3 and caspase-12. Additionally, Tauroursodeoxycholate inhibits ERK.

Tauroursodeoxycholate-D4-1 is the deuterated form of Tauroursodeoxycholate. Tauroursodeoxycholate (T2532) (Tauroursodeoxycholic acid) acts as an endoplasmic reticulum stress inhibitor. It significantly reduces the expression of apoptotic molecules such as caspase-3 and caspase-12, and it also inhibits ERK.

Tauroursodeoxycholate dihydrate (Standard) is the standard substance of Tauroursodeoxycholate dihydrate, and it is applicable for quantitative analysis, quality control, and related research in biochemical experiments. Tauroursodeoxycholate (TUDCA ,Taurolite) dihydrate is a water-soluble bile acid with endoplasmic reticulum stress inhibitory, mitochondrial stabilizing, and anti-apoptotic effects, which significantly reduces the expression of apoptotic molecules, such as Caspase-3 and Caspase-12, and inhibits ERK, reducing endoplasmic reticulum stress mediated cell death.

Tauroursodeoxycholate sodium (Standard) is a reference standard for research and analysis in studies involving Tauroursodeoxycholate sodium. Tauroursodeoxycholate sodium (TUDC) is an endoplasmic reticulum (ER) stress inhibitor, used for the treatment of gallstones and liver cirrhosis.

Tauroursodeoxycholate-D4 Sodium (Standard) is a reference standard of Tauroursodeoxycholate-D4 Sodium intended for quantitative analysis, quality control, and related biochemical research applications. Tauroursodeoxycholate-d4 (sodium) is the deuterated form of Tauroursodeoxycholate sodium. Tauroursodeoxycholate (Tauroursodeoxycholic acid; TUDCA) sodium functions as an endoplasmic reticulum stress inhibitor. It significantly reduces the expression of apoptotic molecules such as caspase-3 and caspase-12. Additionally, Tauroursodeoxycholate inhibits ERK.

Ursodeoxycholic acid-D5 is the deuterated form of Ursodeoxycholic acid. Ursodeoxycholic acid (T0700) (Ursodeoxycholate) is a secondary bile acid produced through the transformation of (cheno)deoxycholic acid by intestinal bacteria. It plays a crucial role in maintaining intestinal barrier integrity and is essential for lipid metabolism. As a signaling molecule, Ursodeoxycholic acid (T0700) functions by interacting with bile acid-activated receptors, including the G protein-coupled bile acid receptor 5 (TGR5, GPCR19) and the Farnesoid X receptor (FXR). It is used in research related to various liver and gastrointestinal disorders and is effective when administered orally.