acetylcysteine

Acetylcysteine (NAC) is an N-acetyl derivative of cysteine, a ROS inhibitor and mucolytic agent. Acetylcysteine induces apoptosis, can be used to reduce mucus thickness, and has anti-influenza viral activity.

N-Acetylcysteine lysinate is a bioactive chemical.

Acetylcysteine-CoA (Acetylcysteine-coenzyme A) is a derivative of coenzyme A.

N-Acetylcysteine amide is a thiol antioxidant and a neuroprotective agent with cell permeability and blood-brain barrier permeability. N-Acetylcysteine amide reduces ROS production.

N-Acetyl-L-cysteine ethyl ester (NACET) is a derivative of the amino acid L-cysteine. NACET has been shown to exhibit diverse biochemical and physiological effects, including inhibition of protein synthesis, inhibition of DNA replication, and induction of apoptosis. Additionally, it has demonstrated anti-inflammatory, antioxidant, and anti-cancer activity.

S-Nitroso-N-acetylcysteine inhibits proliferation and survival of multiple MM cells. It acts by the S-nitrosylation-dependent inhibition of STAT3 and NF-κB.

Acetylcysteine-15N is a version of Acetylcysteine labeled with nitrogen-15 (\[^15N\]). Acetylcysteine (N-Acetylcysteine) functions as a mucolytic agent, effectively reducing mucus viscosity. As a reactive oxygen species (ROS) inhibitor, it serves as a cysteine precursor and prevents heme-induced ferroptosis by neutralizing toxic lipids produced via arachidonic acid-dependent 5-lipoxygenase activity. Additionally, Acetylcysteine can induce apoptosis and exhibits antiviral activity against influenza. It represents the most stable form of cysteine for drug delivery and is utilized in studies of disulfidptosis.

Acetylcysteine-d3 is the deuterated form of Acetylcysteine. Acetylcysteine (N-Acetylcysteine), a mucolytic agent, is utilized to decrease mucus viscosity. It functions as an ROS inhibitor and serves as a precursor to cysteine. By neutralizing the toxic lipids produced via arachidonic acid-dependent 5-lipoxygenase activity, it helps prevent ferroptosis induced by heme. Furthermore, Acetylcysteine can trigger apoptosis and exhibits antiviral activity against influenza viruses. It represents the most stable form of cysteine for drug delivery, suitable for disulfidptosis research.

S-Acetyl-cysteine-CoA (S-Acetyl-cysteine-coenzyme A) is a derivative of coenzyme A.

N,N'-Diacetyl-L-cystine (DiNAC) is the disulphide dimer of N-acetylcysteine with immunomodulating properties. N,N'-diacetyl-L-cystine also has antiatherosclerotic effects in Watanabe-heritable hyperlipidemic rabbit (WHHL) rabbits. N,N'-diacetyl-L-cystine is also an effective and orally active modulator of contact sensitivity/delayed-type hypersensitivity reactions in rodents.

1,4-Anthraquinone is an anticancer drug that blocks nucleoside transport, inhibits macromolecule synthesis, induces DNA fragmentation, and decreases the growth and viability of L1210 leukemic cells in the same nanomolar range as daunorubicin in vitro.1,4-

I-152 is a coupling of N-acetylcysteine and β-mercaptoethylamine. I-152 has antiproliferative, antiviral, and immunomodulatory activities. I-152 is used in the study of viral infections because it activates NRF2 and ATF4 signaling.

Nesosteine sodium salt is an effective bronchitis inhibitor that enhances the rheology and production of bronchial mucus in rabbits. When administered via sulfuric acid nebulization in animals with induced bronchitis, this compound significantly reduces the viscosity of bronchial mucus and increases mucus production in healthy animals. It exhibits greater activity compared to other mucoregulatory drugs, such as Sobrerol, N-acetylcysteine, bromhexine, ambroxol, S-carboxymethylcysteine, and thiopropamine. Additionally, Nesosteine sodium salt reduces the total protein content in the airway bronchial mucus, including albumins and mucoproteins α1, α2, β, and γ.