fusidic acid

Fusidic acid (Fusidine) is an antibiotic isolated from the fermentation broth of Fusidium coccineum.

Fusidic acid sodium salt (SQ-16360) is a sodium salt form of fusidic acid, a bacteriostatic antibiotic derived from the fungus Fusidium coccineum and used as a topical medication to treat skin infections.

3-keto Fusidic acid (3-keto FA) is an active metabolite of the antibiotic fusidic acid .13-keto FA is active againstM. tuberculosis(MIC99= 1.25 μM).

11-Keto fusidic acid exhibits potent antibacterial efficacy against Staphylococcus aureus, demonstrating a minimum inhibitory concentration (MIC) of 0.078 μg/mL.

Lumifusidic Acid, a derivative of Fusidic Acid, is classified as a static antibiotic. Fusidic Acid inhibits protein synthesis in prokaryotes by blocking the ribosome-dependent activity of the G factor and the translocation of peptidyl-tRNA. Additionally, this compound can inhibit the lytic effect of nitric oxide on islet cells.

Fusidic acid-D6 is the deuterated form of Fusidic acid. Fusidic acid (T1160) (Fusidate) is a steroidal antibacterial antibiotic derived from Fusidium coccineum, lacking corticosteroid effects. It inhibits bacterial growth by preventing the ribosome from releasing elongation factor G (EF-G) during protein synthesis.

Fusidic Acid (Standard) is the standard substance of Fusidic Acid, and it is applicable for quantitative analysis, quality control, and related research in biochemical experiments. Fusidic acid (Fusidine) is an antibiotic isolated from the fermentation broth of Fusidium coccineum.

Fusidic acid acyl β-D-glucuronide is a reagent used in biochemical reactions.

Diethanolamine Fusidate is a bacteriostatic antibiotic with similar activity and better absorption after oral administration (in animals) than the sodium salt of Fusidic Acid. This product inhibits protein synthesis in prokaryotes by inhibiting the ribosome-dependent activity of G factor and translocation of peptidyl-tRNA.

Anti-inflammatory agent 50 (compound a1), a derivative of Fusidic acid, exerts its effects through inhibition of inflammatory mediators including NO, IL-6, and TNF-α. It mitigates acute lung injury by modulating these mediators and suppressing the MAPK, NF-κB, and NLRP3 inflammasome signaling pathways [1].