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Farnesyl pyrophosphate (Farnesyl diphosphate), a 15-carbon isoprenoid from the mevalonate (MVA) pathway, functions as a metabolic intermediary and TRPM2 (TRP Channel) agonist, facilitating TRPM2 channel opening and ion influx. It is crucial in synthesizing cholesterol, ubiquinones, and geranyl-geranyl pyrophosphate (GGPP), and in protein farnesylation decoration [1].

Farnesyl pyrophosphate-D2 is the deuterium-labeled version of Farnesyl pyrophosphate (T73755) triammonium. This compound, also known as Farnesyl pyrophosphate (T73755) ammonium salt, is a 15-carbon isoprenoid and a metabolic intermediate in the mevalonate (MVA) pathway. It acts as an agonist of TRPM2 (TRP Channel), enabling the channel to open and allowing ion flow into cells. Additionally, Farnesyl pyrophosphate (T73755) ammonium salt serves as a crucial branching substrate for cholesterol synthesis, ubiquinone synthesis, protein prenylation, and the synthesis of geranylgeranyl pyrophosphate (GGPP).

Farnesyl pyrophosphate-D2 triammonium is a deuterium-labeled variant of farnesyl pyrophosphate triammonium. This 15-carbon isoprenoid serves as a metabolic intermediate in the mevalonate (MVA) pathway. Additionally, farnesyl pyrophosphate ammonium salt acts as an agonist for the TRPM2 (TRP Channel), facilitating ion entry into cells. It is also a crucial substrate for cholesterol synthesis, ubiquinone synthesis, protein farnesylation, and geranylgeranyl pyrophosphate (GGPP) production.

Farnesyl Pyrophosphate ammonium salt is an isomer of (E,E)-Farnesyl Pyrophosphate ammonium salt, which is a key metabolic intermediate in the mevalonate (MVA) pathway and an agonist of TRPM2, and is involved in cholesterol synthesis, ubiquinone synthesis, protein farnesylation and other physiological processes.

Farnesyl Pyrophosphate ammonium (Farnesyl diphosphate ammonium) is a metabolic intermediate of the MVA pathway that acts as a newly identified danger signal to trigger acute cell death and induce neuronal loss in stroke.