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Pancreatitis, including both acute and chronic forms, involves complex pathogenic mechanisms such as premature activation of pancreatic enzymes, release of inflammatory cytokines, and oxidative stress. Commonly used animal models include L-arginine induction, bile duct ligation, and alcohol-induced methods. These models are valuable for investigating pancreatic tissue injury mechanisms, evaluating anti-inflammatory therapies, and exploring intervention strategies for multiple organ dysfunction.
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