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Results for "

T8003

" in TargetMol Product Catalog
  • Inhibitors & Agonists
    9
    TargetMol | All_Pathways
  • Peptide Products
    8
    TargetMol | Peptide_Products
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    1
    TargetMol | PROTAC
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    1
    TargetMol | All_Pathways
Methotrexate disodium
T80037413-34-5
Methotrexate disodium is an tetrahydrofolate dehydrogenase inhibitor
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Oxyntomodulin (bovine, porcine) (TFA)
Glucagon-37 (bovine, porcine) (TFA)
T80030
Oxyntomodulin (bovine, porcine) TFA is a 37-amino acid peptide hormone that functions as a glucagon-like peptide 1 (GLP-1) receptor agonist [1].
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[D-Pro2] Spantide I TFA
T80031
[D-Pro2] Spantide I TFA is an analog of Spantide I, serving as a selective antagonist of the NK1 receptor [1].
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FITC-α-Bungarotoxin
T80032
FITC-α-Bungarotoxin is the FITC-labeled form of α-Bungarotoxin, which acts as a competitive antagonist at nicotinic acetylcholine receptors (nAChRs) [1].
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Biotin-α-Bungarotoxin
T80033
Biotin-α-Bungarotoxin is a biotinylated form of α-Bungarotoxin, which acts as a competitive antagonist at nicotinic acetylcholine receptors (nAChRs) [1].
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Margatoxin TFA
T80034
Margatoxin TFA, an alpha-KTx scorpion toxin isolated from Centruroides margaritatus venom, is a 39-amino-acid peptide that serves as a high-affinity inhibitor of Kv1.3 (Kd = 11.7 pM). It also inhibits Kv1.2 (Kd = 6.4 pM) and Kv1.1 (Kd = 4.2 nM), making it a valuable tool for ion channel research [1] [2].
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Biotin-β-Amyloid (1-42), human TFA
Biotin-amyloid β-peptide (1-42) (human) TFA
T80035
Biotin-β-Amyloid (1-42), human TFA, also known as Biotin-Amyloid β-Peptide (1-42) (human) TFA, is a biotin-labeled 42-amino acid peptide implicated in the pathogenesis of Alzheimer's disease.
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TargetMol | Citations Cited
AmmTX3 TFA
T80036
AmmTX3 TFA, a peptide toxin derived from Androctonus mauretanicus scorpion venom, functions as a selective blocker of the K_v4 channel. It inhibits the A-type K^+ current with an inhibition constant (K_i) of 131 nM [1] [2].
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ATX-II
T8003960748-45-0
ATX-II, an Na+ channel modulator toxin derived from the sea anemone (Anemonia sulcata), causes delayed inactivation of the Na+ channels and has been linked to pulmonary vein arrhythmogenesis and atrial fibrillation [1] [2]. It exhibits interaction with halothane, caffeine, and ryanodine.
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