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By Target- PROTAC Linker
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Results for "
cl-2 linker
" in TargetMol Product Catalog- Inhibitors & Agonists9
- PROTAC Products8
CL2 Linker
T177302270986-66-6
CL2 Linker is a cleavableADC linker. CL2-SN-38 and CL2A-SN-38 are equivalent in drug substitution (~6), cell binding (Kd ~1.2 nM), cytotoxicity (IC50 ~2.2 nM), and serum stability in vitro (t1 2 ~20 hours)[1][2].
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(S,R,S)-AHPC-PEG2-C4-Cl
E3 ligase Ligand-Linker Conjugates 10, VHL Ligand-Linker Conjugates 7, VH032-PEG2-C4-Cl
T179101835705-57-1
(S,R,S)-AHPC-PEG2-C4-Cl is a small molecule HaloPROTAC, comprising the (S,R,S)-AHPC based VHL ligand and a 2-unit PEG linker, that induces the degradation of GFP-HaloTag7 in cell-based assays[1].
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(S,R,S)-AHPC-(C3-PEG)2-C6-Cl
VHL Ligand-Linker Conjugates 11, E3 ligase Ligand-Linker Conjugates 11
T179121835705-61-7
(S,R,S)-AHPC-(C3-PEG)2-C6-Cl is a small molecule HaloPROTAC incorporating the (S,R,S)-AHPC-based VHL ligand and a 2-unit PEG linker, capable of inducing degradation of GFP-HaloTag7 in cell-based assays [1].
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VH032-PEG5-C6-Cl
HaloPROTAC 2
T179961799506-06-1
VH032-PEG5-C6-Cl (HaloPROTAC 2) is a small molecule that includes the VH032-based VHL ligand and a 5-unit PEG linker, capable of inducing the degradation of GFP-HaloTag7 in cell-based assays[1].
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Boc-C5-O-C5-O-C6-Cl
PROTAC Linker 2
T186381835705-52-6
Boc-C5-O-C5-O-C6-Cl (PROTAC Linker 2) is a chemical compound primarily used as a linker in PROTAC applications, facilitating the connection between a specific tyrosine kinase inhibitor (TKI) and the E3 recruiting ligand.
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trans-(S,R,S)-AHPC-Me-Amide-cyclohexane-pyrimidine-Cl
T201567
trans-(S,R,S)-AHPC-Me-Amide-cyclohexane-pyrimidine-Cl is an E3 ligase ligand-Linker conjugate that is utilized in the synthesis of PROTACs, such as PROTAC BRM BRG1 degrader-2.
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Azido-PEG2-C6-Cl
T402012568146-55-2
Azido-PEG2-C6-Cl is a PROTAC linker belonging to the PEG class and can be used to synthesize PROTAC molecules.
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NH2-PEG2-C6-Cl
NH2-PEG2-C6-Cl
T40858744203-60-9
NH2-PEG2-C6-Cl is a PEG-based linker for PROTACs, facilitating the formation of PROTAC molecules by joining two essential ligands. It enables selective protein degradation by leveraging the ubiquitin-proteasome system within cells.
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