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citrulline malate

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Citrulline malate
Stimol, L-Citrulline DL-malate
T2020070796-17-7
Citrulline malate is a cpd of anti-fatigue.
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L-Citrulline
T4813372-75-8
L-Citrulline, an amino acid, is derived from ornithine during the catabolism of proline, glutamate, or glutamine, and also from L-arginine via the arginine-citrulline pathway.
  • $42
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L-Citrulline-13C
TMID-113194740-46-2
L-Citrulline-13C is L-Citrulline labeled with 13C. This amino acid is derived from the breakdown of ornithine, which originates from the catabolism of proline, glutamine, and glutamate salts, or it can be obtained from l-arginine through the arginine-citrulline pathway.
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L-Citrulline-d7
TMID-12022483831-24-7
L-Citrulline-d7 is the deuterated form of L-Citrulline. L-Citrulline is an amino acid derived from the catabolism of ornithine, which stems from proline, glutamine, and glutamate, or it can be obtained through the arginine-citrulline pathway from l-arginine.
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2-Amino-5-ureidopentanoic acid (Standard)
TMSM-3319627-77-0
2-Amino-5-ureidopentanoic acid (Standard) is a reference standard for research and analysis in studies involving 2-Amino-5-ureidopentanoic acid. 2-Amino-5-ureidopentanoic acid (Citrulline) exists in the liver, is not a protein component, is an important intermediate in the human urea cycle, can be combined with ornithine, arginine to treat hyperammonemia.
  • $98
4-6 weeks
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2-Amino-5-ureidopentanoic acid
DL-Citrulline, Citrulline
T2931627-77-0
2-Amino-5-ureidopentanoic acid (Citrulline) exists in the liver, is not a protein component, is an important intermediate in the human urea cycle, can be combined with ornithine, arginine to treat hyperammonemia.
  • $40
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L-Citrulline-d6
TMID-06711331908-61-2
L-Citrulline-d6 is the deuterated form of L-Citrulline. L-Citrulline is an amino acid derived from ornithine, which results from the catabolism of proline, glutamine, and glutamate, or obtained through the arginine-citrulline pathway involving l-arginine.
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D-Citrulline
H-D-Cit-OH
T20034013594-51-9
D-Citrulline (H-D-Cit-OH), a stereoisomer of L-citrulline, effectively reduces cardiac contractile dysfunction caused by polymorphonuclear leukocyte (PMN) in isolated perfused rat hearts undergoing ischemia/reperfusion. This protective effect is mediated through a non-NO-mediated mechanism.
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10-14 weeks
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L-Citrulline-d4
TMID-07841217474-00-4
L-Citrulline-d4 is the deuterated form of L-Citrulline. L-Citrulline, an amino acid, is derived from the catabolic processes involving ornithine, originating from the breakdown of proline, glutamine, and glutamates, or it can be obtained through the l-arginine pathway known as the arginine-citrulline cycle.
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L-Citrulline (Standard)
TMIM-00053372-75-8
L-Citrulline (Standard) is available for quantitative analysis.) L-Citrulline is a non-essential amino acid involved in the nitric oxide cycle and the urea cycle, promotes vasodilatation, has antioxidant and anti-inflammatory properties, is orally available, and crosses the blood-brain barrier through LAT1. It is an agonist of eNOS (endothelial nitric oxide synthase), iNOS (inducible nitric oxide synthase), and mTORC1 (rapamycin targeting protein complex 1), and an inhibitor of SREBP-1 (sterol regulatory element binding protein 1).
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