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Adiponectin Protein, Human, Recombinant (hFc) V2

TargetMol | SPR
Catalog No. TMPY-02848U

Adiponectin Protein, Human, Recombinant (hFc) V2 is expressed in HEK293 Cells. The accession number is Q15848.

Adiponectin Protein, Human, Recombinant (hFc) V2

Adiponectin Protein, Human, Recombinant (hFc) V2

TargetMol | SPR
Catalog No. TMPY-02848U
Adiponectin Protein, Human, Recombinant (hFc) V2 is expressed in HEK293 Cells. The accession number is Q15848.
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Product Information

Biological Activity
Activity has not been tested. It is theoretically active, but we cannot guarantee it.
Description
Adiponectin Protein, Human, Recombinant (hFc) V2 is expressed in HEK293 Cells. The accession number is Q15848.
Species
Human
Expression System
HEK293 Cells
TagC-hFc
Accession NumberQ15848
Synonyms
GBP28,APM-1,APM1,ADPN,ADIPQTL1,adiponectin, C1Q and collagen domain containing,Adiponectin,ACRP30,ACDC
Construction
Glu19-Asn244
Protein Purity
> 95% as determined by Tris-Bis PAGE; > 95% as determined by HPLC
Molecular Weight51.2 kDa (Predicted); 52-70 kDa (Due to glycosylation)
Endotoxin< 1.0 EU/μg of the protein as determined by the LAL method.
FormulationLyophilized from 0.22 μm filtered solution in PBS (pH 7.4). Normally 8% trehalose is added as protectant before lyophilization.
Stability & Storage
Lyophilized powders can be stably stored for over 12 months, while liquid products can be stored for 6-12 months at -80°C. For reconstituted protein solutions, the solution can be stored at -20°C to -80°C for at least 3 months. Please avoid multiple freeze-thaw cycles and store products in aliquots.
ShippingIn general, Lyophilized powders are shipping with blue ice. Solutions are shipping with dry ice.
Research Background
Adiponectin, also known as Acrp30, is an adipocyte-derived protein with wide ranging paracrine and endocrine effects on metabolism and inflammation.Important adipokine involved in the control of fat metabolism and insulin sensitivity, with direct anti-diabetic, anti-atherogenic and anti-inflammatory activities. Stimulates AMPK phosphorylation and activation in the liver and the skeletal muscle, enhancing glucose utilization and fatty-acid combustion. Antagonizes TNF-alpha by negatively regulating its expression in various tissues such as liver and macrophages, and also by counteracting its effects.

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